Dual Inhibition of Mycobacterium tuberculosis and the Host TGFBR1 by an Anilinoquinazoline. Read more about Dual Inhibition of Mycobacterium tuberculosis and the Host TGFBR1 by an Anilinoquinazoline.
Fused Tetrahydroquinolines Are Interfering with Your Assay. Read more about Fused Tetrahydroquinolines Are Interfering with Your Assay.
Optimization of 3-Cyano-7-cyclopropylamino-pyrazolo[1,5-a]pyrimidines toward the Development of an In Vivo Chemical Probe for CSNK2A. Read more about Optimization of 3-Cyano-7-cyclopropylamino-pyrazolo[1,5-a]pyrimidines toward the Development of an In Vivo Chemical Probe for CSNK2A.
Chemical proteomics reveals the target landscape of 1,000 kinase inhibitors. Read more about Chemical proteomics reveals the target landscape of 1,000 kinase inhibitors.
Open science discovery of potent noncovalent SARS-CoV-2 main protease inhibitors. Read more about Open science discovery of potent noncovalent SARS-CoV-2 main protease inhibitors.
The PB1 and the ZZ domain of the autophagy receptor p62/SQSTM1 regulate the interaction of p62/SQSTM1 with the autophagosome protein LC3B. Read more about The PB1 and the ZZ domain of the autophagy receptor p62/SQSTM1 regulate the interaction of p62/SQSTM1 with the autophagosome protein LC3B.
Open Source Antibiotics: Simple Diarylimidazoles Are Potent against Methicillin-Resistant Staphylococcus aureus. Read more about Open Source Antibiotics: Simple Diarylimidazoles Are Potent against Methicillin-Resistant Staphylococcus aureus.
Scaling of an antibody validation procedure enables quantification of antibody performance in major research applications. Read more about Scaling of an antibody validation procedure enables quantification of antibody performance in major research applications.
Discovery of a First-in-Class Small-Molecule Ligand for WDR91 Using DNA-Encoded Chemical Library Selection Followed by Machine Learning. Read more about Discovery of a First-in-Class Small-Molecule Ligand for WDR91 Using DNA-Encoded Chemical Library Selection Followed by Machine Learning.
An atypical GABARAP binding module drives the pro-autophagic potential of the AML-associated NPM1c variant. Read more about An atypical GABARAP binding module drives the pro-autophagic potential of the AML-associated NPM1c variant.
