Benzo-isoquinoline-diones As Potent and Selective Inhibitors of BRPF2 and TAF1/TAF1L Bromodomains. Read more about Benzo-isoquinoline-diones As Potent and Selective Inhibitors of BRPF2 and TAF1/TAF1L Bromodomains.
The RNF168 paralog RNF169 defines a new class of ubiquitylated-histone reader involved in the response to DNA damage. Read more about The RNF168 paralog RNF169 defines a new class of ubiquitylated-histone reader involved in the response to DNA damage.
A multi-crystal method for extracting obscured crystallographic states from conventionally uninterpretable electron density. Read more about A multi-crystal method for extracting obscured crystallographic states from conventionally uninterpretable electron density.
Global survey of the immunomodulatory potential of common drugs. Read more about Global survey of the immunomodulatory potential of common drugs.
Hyperactive locomotion in a Drosophila model is a functional readout for the synaptic abnormalities underlying fragile X syndrome. Read more about Hyperactive locomotion in a Drosophila model is a functional readout for the synaptic abnormalities underlying fragile X syndrome.
Recombinant Protein Expression in E. coli : A Historical Perspective. Read more about Recombinant Protein Expression in E. coli : A Historical Perspective.
N- and C-Terminal Truncations to Enhance Protein Solubility and Crystallization: Predicting Protein Domain Boundaries with Bioinformatics Tools. Read more about N- and C-Terminal Truncations to Enhance Protein Solubility and Crystallization: Predicting Protein Domain Boundaries with Bioinformatics Tools.
In Vivo Biotinylation of Antigens in E. coli. Read more about In Vivo Biotinylation of Antigens in E. coli.
A specific and covalent JNK-1 ligand selected from an encoded self-assembling chemical library. Read more about A specific and covalent JNK-1 ligand selected from an encoded self-assembling chemical library.
Protein destabilization and loss of protein-protein interaction are fundamental mechanisms in cblA-type methylmalonic aciduria. Read more about Protein destabilization and loss of protein-protein interaction are fundamental mechanisms in cblA-type methylmalonic aciduria.
